Stem cell researchers have uncovered for the first time why adult human cardiac myocytes have lost their ability to proliferate, perhaps explaining why the human heart has little regenerative capacity ...

Mst1 causes the transport of FoxO1 from the cell cytoplasm to the nucleus by phosphorylating it. This is shown by the colocalization of the green and the blue stain when Ad-Mst1 is added. Researchers ...

The scarring of the heart tissue after a myocardial infarction suggests that the myocytes are unable to divide. In June 7 New England Journal of Medicine Antonio Beltrami and colleagues from the New ...

Expression of Concern: Beltrami AP et al. Evidence That Human Cardiac Myocytes Divide after Myocardial Infarction. N Engl J Med 2001;344:1750-7 and Quaini F et al. Chimerism of the Transplanted Heart.

Proceedings of the National Academy of Sciences of the United States of America, Vol. 114, No. 3 (January 17, 2017), pp. E270-E279 (10 pages) Cardiac myocytes normally initiate action potentials in ...

bit.bio has today launched a new product, ioSkeletal Myocytes. It’s the first product of its kind that provides physiologically relevant human skeletal muscle cells that are reliable, reproducible and ...

The following studies were undertaken to develop a cultured uterine myocyte model which would allow further clarification of the adrenergic signal transduction mechanisms utilized by these myocytes.

CAMBRIDGE, England--(BUSINESS WIRE)--#Celltherapy--bit.bio, the company coding human cells for novel cures, today launches its first set of muscle cell disease model products designed to advance the ...

In a recent study published in the journal PNAS, researchers examined the relationship between cell size and count across the human body, establishing a quantitative framework and uncovering ...